We tried to demonstrate such an increase by analysing the levels of PGE2-G in the malonate-lesioned striatum, and, particularly, after the inhibition of MAGL, which should facilitate the formation of PGE2-G (see below)

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We tried to demonstrate such an increase by analysing the levels of PGE2-G in the malonate-lesioned striatum, and, particularly, after the inhibition of MAGL, which should facilitate the formation of PGE2-G (see below). striatum 24?h after the lesion simultaneously with additional pro-inflammatory reactions. The COX-2-derived 2-AG metabolite, prostaglandin E2 glyceryl Continue Reading